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In this review I aim to re-examine the contested concept of food addiction as a disease in the context of emerging GLP-1based obesity pharmacotherapies, integrating perspectives from neurobiology, societal impacts, and early evidence for their potential application in other substance-use disorders

Recombinant human KAI1/CD82 attenuates M1 macrophage polarization on LPS-stimulated RAW264.7 cells via blocking TLR4/JNK/NF-B signal pathway

GSH production in tumours is dispensable GSH facilitates tumorigenesis 17,18,19

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