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Employing gene editing techniques alongside both in vivo and in vitro experiments, we provide novel evidence that the dysfunction of SLC25A39-mediated mitochondrial import of GSH constitutes a pathological alteration following TBI, leading to mitochondrial dysfunction and subsequent initiation of oxidative stress and neuronal cell death

It acts through the activation of receptors present on primary afferents

Taking it alongside prescription blood thinners such as Warfarin, Clopidogrel, or even daily over-the-counter aspirin can increase the risk of bruising and prolonged bleeding

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