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Strawn JR, Chu WJ, Whitsel RM, Weber WA, Norris MM, Adler CM, et al

Priorities include defining the in situ biochemical flux from lactate to lactylating capacity using isotope tracing, mapping when lactylation is reversible during therapy, and testing whether CNS lactate shuttling linked to neural activity shapes tumor and myeloid lactylation states

As a critical molecule in cellular energy metabolism and DNA repair, insufficient NAD+ directly triggers a series of metabolic abnormalities, including impaired mitochondrial function, reduced energy metabolism efficiency, and weakened DNA repair capacity

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