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After ischemic injury, microglia, as the intrinsic immune cells of the central nervous system, can be rapidly activated by ATP released from damaged neurons and surrounding glial cells via the P2X7 receptor pathway, thereby promoting the synthesis and secretion of pro-inflammatory factors (35)

compete with acetylcholine (ACh) for binding to postsynaptic nicotinic receptor sites SECONDARY MECHANISM: Nondepolarizing relaxants also from motor nerve endings by still poorly understood mechanism(s)

10.1038/s41419-020-2334-2 114 YanH.HuangZ.BaiQ.ShengY.HaoZ.WangZ.et al (2018)