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Specifically, the study found ARGX-113 to significantly increase FcRn levels both in human cell lines modified to overexpress FcRn and in human cells with endogenous expression of the receptor

This dysfunction manifests as reduced vascular response to dilators (particularly to NO and prostacyclin), heightened pro-inflammatory responses leading to vascular wall inflammation, increased oxidative stress contributing to vascular damage, and pathological changes

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[Google Scholar] 50.Di Pietrantonj C, Rivetti A, Marchione P, Debalini MG, Demicheli V